Neutrophil Extracellular Traps (NETs) are potentially of interest to us. This image from a 2020 paper by a Chinese team (Neutrophil Extracellular Traps: Signaling Properties and Disease Relevance Tiewei Li, Zhengyan Zhang, Xiaojuan Li, Geng Dong, Min Zhang, Zhe Xu, and Junmei Yang) gives a feel for what is going on. Neutrophils are a type of white blood cell; they are part of the immune system. When activated, they throw out fibres of DNA, comprised of histone and cytoplasmic granule proteins. The fibres can capture pathogens, degrade toxins and kill bacteria. And they can provide scaffolding for protein and cell binding and can help in blood clotting. But, like a lot of the immune system it seems, things can go wrong and there can be collateral damage. Tissues nearby can be damaged due to the proteins carried (as is the case in the picture, with damage to the endothelium) or clotting can happen where it isn't helpful. It's being suggested that excessive formation of these NETs can lead to a range of diseases (like autoimmune diseases, sepsis, thrombosis and atherosclerosis). And that managing the formation of NETs could prevent or treat quite a number of health conditions.