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What could be the mechanism behind virus latency?

Discussion in 'Infections: Lyme, Candida, EBV ...' started by AllenJB, Nov 3, 2019.

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  1. AllenJB

    AllenJB Established Member (Voting Rights)

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    You can't feel cells dying off. You can only feel nerves with intact connections to the brain. Apoptosis releases histamine and a cytokine response that causes pain in the nerves in that area. You can detect histamine response as rhinitis. Completed nerve death is perceived as numbness. I guess it's because there are a lot of cells dying off at once which is greater than the usual slow process. When this really gets going I have to use naproxen sodium to counter the cytokines, benadryl for the histamine and gabapentin for nerve pain (add acetaminophen when particularly bad).

    How do I know? The process sure acts like apoptosis. It can be inhibited by strong antioxidants or stopping the pro-apoptosis supplements. It leaves scar tissue behind. It improves Raynaud's disease temperature changes and reverses post-ME cold skin areas. Etc.

    To measure apoptosis:

    "In apoptosis assays, DNA fragmentation and sub-G1 phase populations were increased, and the mitochondrial membrane potential decreased significantly after treatments." https://www.ncbi.nlm.nih.gov/pubmed/26327534

    So it looks like one needs to look at the cells microscopically. I would biopsy musculoskeletal cells in asyptomatic tender, weak muscles. Fascia, muscle and tendons. Test pretreatment reactiveness then re-biopsy post treatment and look for changes.
     
    Last edited: Nov 5, 2019

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