BPS attempts at psychologizing Long Covid

Barry said:
Hopefully there will be much more widespread motivation and resources to get to the bottom of long covid than there ever has been for ME, so that some real answers are found. In the process it would be great if it furthered better understanding of ME/CFS.
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We may end up there by different pathways but there’s often some kind of connection.
 
Kalliope said:
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Ah yes, Mr "Historian of medicine" out there pretending the lack of an association with acute disease severity means the psychosomatic model is correct, even though it has always, ALWAYS, been argued that it must be one of the main predictors, essentially the scary event that causes all the panic. I mean seriously this was argued ad nauseam and here they just do a full 180 spin as if they never said any of this. Of course when inconvenient the opposite has also often been argued but always going back and forth depending on what's convenient.

This thing where "experts" are allowed to endlessly speculate and bullshit, oftentimes completely reversing their position on some fundamental aspects without ever changing any of the consequences they have endlessly argued about, is really not working out and really needs to stop. None of this is normal. In any other field of expertise such people would rightfully considered to be cranks without any credibility.

NONE of this is normal. This is the exact opposite of what expertise is supposed to be about.
 
AR68 said:
Mentioned earlier the year 1988. This happens in the same year....
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It's weird reading this and reconciling with some people trying to distance ME from LC by saying shortness of breath isn't a symptom of ME. Even more weird reconciling the fact that the very same people dismissing the hyperventilation here have always argued that the only symptom of ME is fatigue.

Almost like they're completely full of it. Almost.
 
Snow Leopard said:
Is there any proof of this, or is this just speculation based on coincidence?
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In my experience these sort of folk operate on the principle of plausible deniability, and operating well below the radar. It is their modus operandi. Given what we know of their history, the likelihood of them not "reaching out" as it suits them seem pretty implausible to me. But I grant you, that is hardly evidence.
 
Snow Leopard said:
Is there any proof of this, or is this just speculation based on coincidence?
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Just an educated guess - based on known associations between the Guardian, SMC, SW & Goldacre. But this really does belong in the "Who is SW?" thread.
Barry said:
plausible deniability, and operating well below the radar. It is their modus operandi.
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Macavity, Macavity, there’s no one like Macavity,
There never was a Cat of such deceitfulness and suavity.
He always has an alibi, and one or two to spare:
At whatever time the deed took place—MACAVITY WASN’T THERE!
 
During the epidemics people who were active were more likely to get ME, for instance in a convent that was also a teacher training college, the pupils got ME, the nuns did not. In polio activity was associated with symptoms too.

It is possible that people with mild disease get LC because they stay active, not saying it is true but it is a possible explanation that does not involve anything psychosomatic.

Some of what they are saying sounds like a moral judgement. Heroes get really sick but losers who are looking for attention exaggerate their symptoms and feel sorry for themselves.
 
Here is one explanation:

Abstract
The worldwide spread of a novel influenza A (H1N1) virus in 2009 showed that influenza remains a significant health threat, even for individuals in the prime of life. This paper focuses on the unusually high young adult mortality observed during the Spanish flu pandemic of 1918. Using historical records from Canada and the U.S., we report a peak of mortality at the exact age of 28 during the pandemic and argue that this increased mortality resulted from an early life exposure to influenza during the previous Russian flu pandemic of 1889-90. We posit that in specific instances, development of immunological memory to an influenza virus strain in early life may lead to a dysregulated immune response to antigenically novel strains encountered in later life, thereby increasing the risk of death. Exposure during critical periods of development could also create holes in the T cell repertoire and impair fetal maturation in general, thereby increasing mortality from infectious diseases later in life. Knowledge of the age-pattern of susceptibility to mortality from influenza could improve crisis management during future influenza pandemics.
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IAPT Post-COVID Syndrome Training Webinars
We’re pleased to share an invite to an upcoming “long COVID” training webinar series hosted by our national Improving Access to Psychological Therapies (IAPT) Programme team.

Building on the weekly webinar series held during the lockdown earlier this summer, the team is hosting a five part clinical webinar series on the role (IAPT) can play in supporting people who have had COVID and, in particular, those that are experiencing persistent symptoms, which are now commonly being referred to as “long COVID” and “post-COVID syndrome”. In addition to the delivery of IAPT interventions, they’ll bring a range of physical health experts into each session as well as lived experience from people recovering from COVID. The primary audience for each session is listed below, but we would welcome others who may find the sessions helpful to their practice (both therapists and clinical/service leads in IAPT, in addition to others in the wider system, including commissioners, and physical health services supporting people with post-COVID syndrome). We recommend that anyone interested in the series attends the first session which will set important context and background for the following sessions.
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https://cptraininghub.nhs.uk/event/iapt-post-covid-syndrome-training-webinars/
 
...they’ll bring a range of physical health experts into each session as well as lived experience from people recovering from COVID.

What could go wrong with that? I would have thought that those who recovered are not actually a model for those who don't. Useful for a research compare and contrast, but not as clinical model for all COVID patients to follow.
 
Andy said:
IAPT Post-COVID Syndrome Training Webinars

https://cptraininghub.nhs.uk/event/iapt-post-covid-syndrome-training-webinars/
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Using low intensity interventions to help people recover after COVID
Tuesday 12 January 2021, 1pm – 3pm

Primary audience: IAPT therapists, clinical supervisors and managers, physical health practitioners (physiotherapists, occupational therapists and doctors working with people recovering from COVID)

This webinar will focus on using low intensity psychoeducational IAPT interventions for people recovering after COVID and will include specialist Step 2 interventions that might be required to support people with post-COVID syndrome / long COVID e.g. breathlessness. This session will also introduce broader psychoeducational interventions including the Your Covid Recovery platform and the role that exercise and activity management can play.
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As psychoeducational programmes have such an impressive impact on other post-viral conditions they assume it will work with post-covid?!
 
Sly Saint said:
"
Using low intensity interventions to help people recover after COVID
Tuesday 12 January 2021, 1pm – 3pm"
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Yup. It looks every bit as bad as it sounds. The whole IAPT-LTC program is a complete disaster and of course they want to expand it because the hammer does not distinguish between nails and other objects but once flattened out everything can look like a flattened nail if you want to.
 
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