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Well, it is great for someone looking for an example of how applying the GRADE system can lead to a problematic conclusion. But for the ME/CFS patient community it's highly concerning that proponents of GET have managed to get Gordon Guyat to become involved and sign a statement such as this...

It is not as bad as I thought it would be. I like the sentence: "Do not automatically assume a psychological cause for persistent somatic symptoms in the absence of detectable pathological abnormalities."

Here are some articles about how researchers think there might be a link between ME/CFS and COVID-19 and that this warrants further research: Could the coronavirus trigger post-viral fatigue syndromes? | New Scientist As Their Numbers Grow, COVID-19 “Long Haulers” Stump Experts | Infectious...

This recent letter in JAMA stated: Source: https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2776560

Wessely did an important study where he showed that employment in the Gulf War was associated with increased health complaints that weren't seen in the Bosnia conflict or those military men serving during the Gulf War but not deployed there. It was reported in the Lancet...

Anyone have access to the full-text?

Thanks for explaining it (although I'm not very convinced by the explanation)

EDIT: I thought this was a new paper but it's from 2017! The authors of this paper are NIH-funded researchers who have focused their research on deoxyuridine triphosphate nucleotidohydrolases (dUTPase) encoded by herpesviruses such as Epstein -Barr virus (EBV), human herpesvirus -6 (HHV -6)...

I don't quite see why patients who will later go on to develop long covid or ME/CFS after COVID-19, would have much higher rates of false negatives on PCR testing?

The methods and various pathways are a bit too complex for me to understand but this seems to be the main conclusion/hypothesis behind it:

Thanks for the interesting comments and background. What about diabetes, obesity, heart disease: is there reliable evidence that diet works there as a treatment?

moved thread Ok, but most of these studies are based on confirmed PCR tests which if I understand correctly require virus particles, not necessarily antibodies. On Twitter someone pointed me to this Danish preprint which found that 16% of patients reported fatigue and 13% concentration...

This is the study from the University of Washington that found that only 13.6% of patients reported fatigue and only 2.3% reported brain fog approximately 6 months after confirmed COVID-19 infection. Sequelae in Adults at 6 Months After COVID-19 Infection | Infectious Diseases | JAMA Network...

How come so many ME/CFS papers are published in the Journal of Translational Medicine?

Hope this is the right thread to post this information (feel free to move it moderators) copied to the News from the Netherlands thread.

This post has been copied this thread where it is discussed: Maximal handgrip strength can predict maximal physical performance in patients with chronic fatigue, 2020, Jammes et al

New blog post in which Evelien and I analyze Canadian historian Edward Shorter’s view on ME/CFS. https://mecfsskeptic.com/from-paralysis-to-fatigue-a-critical-review-of-edward-shorters-view-on-chronic-fatigue-syndrome/ Twitter summary:

The general document looks a bit like the rapid NICE guideline. It doesn't say much and doesn't mention the possibility of patients developing ME/CFS. They only mention: The additional document "troubles somatiques fonctionnels" is indeed very problematic. It refers to Fink's bodily distress...