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maybe I am mistaken here, but plex takes out most of your plasma in the body if you are doing a full cycle of treatments,...

@Badpack did you try plasma exchange?

If there is something in serum that causes me, why is plasma exchange not working?

I am of your opinion. Patients and people around patients have to invest more into research themselves. Stop buying shitty supplements and using that money to do research instead would be a good start.

I am very severe, I have been in contact with Dr. Bhupesh, my family has met him. Being a realist, having a little understanding of the complexity of the issue at hand and especially the knowledge that there is so much we do not understand about mitochondria I doubt this will be a breakthrough...

There is a high chance that severe me/cfs patients have a mitochondrial disease.

This is just plain wrong. Your perfectly identified clinical diagnosis of me/cfs still makes you a psychiatric patient in 99% of all cases.

Very interesting and relatively sure a major finding and not a coincidence. ME\CFS is a brainsteminjury. But whst caused it?

This is major, please show to Dr. Davis someone!

This sounds like it could be related to ME. I am only pooing fatty, gooey stuff for years.

they are mofos. want to get cheap and free patients money for a very, very, very low chance of success, it is a win win for Cortene.

not every signal exists for a reason, or for a good reason actually, complex systems fail, all the time, everywhere, if not, you would not get sick first place.

This is what ME is like!

https://www.google.at/amp/s/m.jpost.com/Israel-News/Ben-Gurion-University-develops-miracle-molecule-for-fighting-Lupus-612163/amp

These findings seem a bit weird to me, these are the kind of things we would have found 20 years ago, no? I also remember Dr. Davis saying that when he lowered Th17, some markers actually got worse (gut permeability).

ehhhhhhm, yes, you „use mitochondria to think“.

me/cfs is a mito disorder ... in many but not all me/cfs patients and in in even more people with me/cfs with slow onset and EDS like features, pots and mcad ... just wanted to put it out there and hope I will be still alive when it is proven ... (existing mtdna studies for me/cfs are somewhat...

hey! did you have plasma exchange yet? ty

This is so damn stupid. People with PhDs. It is because me is NOT one disease, but a significant part of the me population has a mitochondrial disorder, be it primary or secondary! You have to take me patients with increased lactate and put them into ONE STUDY GROUP!

(Severe) ME is definitely a mitochondrial disorder, it becomes more and more absurd that nobody is calling it such.