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Thanks for looking more closely at this @Hutan. Maybe I can say it. Believing that you can control the situation has almost zero relationship to the actual outcome. This is in extreme contrast to the general belief of society that we patients are able to achieve a recovery if only we did this...

The responses to this article could reinforce the belief of the investigators that patients are neurotic. Maybe someday they'll consider how it feels for highly stigmatized and neglected patients to have a group of researchers regularly searching for flaws in patients thinking and behaviour...

Why would you have to control for time since symptom onset in a longitudinal study? I don't understand what was done here and why.

Are PVFS and ME/CFS similar entities?

Maybe he thinks PVFS is just fatigue, or he is disagreeing with people who suggested he had PVFS because those people viewed it as fatigue.

I'm wondering if the comment about this not being post-viral fatigue syndrome reveals his own ignorance. He describes how other people misunderstand the illness. He thinks it's clearly not postviral fatigue syndrome. Maybe he says this because there is a mismatch between what he thinks PVFS...

Sounds familiar. Also sounds familiar.

SARS-CoV-2 was circulating in France in late December. https://www.bfmtv.com/sante/coronavirus-le-professeur-cohen-affirme-qu-il-y-avait-un-cas-de-covid-19-en-france-des-le-27-decembre-1906757.html Google translation...

https://www.nap.edu/read/19012/chapter/6#78

Recovery is a mindset and I'm recovered, said the patient from the bed he was confined to. The lightning process practitioner was thrilled she had been able to make the patient recover.

My guess is the decision to conduct a clinical trial of kynurenine means they have some new information.

I don't think they would have a problem claiming that the stress of the pandemic explains development of ME/CFS. It would be very biopsychosocial.

They improvements could disappear in a phase 3 trial, but how does one explain the association between greater improvement and HLA alleles? Is it possible that the treatment is no better than placebo, but the HLA alleles are protective and improve outcomes?

A newspaper is reporting that 61% of the people with suspicious symptoms and/or contact with confirmed cases in two of the hardest hit towns in the Bergamo province had positive serological tests. A second lab found a rate of 59%. At one hospital in Bergamo, the percentage of healthcare workers...

Refugee camps in Europe could be a big problem.

A clear suggestion that there is mass hysteria Of course maybe the higher incidence is down to purely medical reasons or reasons that have nothing to do with hysteria. Like these places having a few doctors that are actually diagnosing the illness.

Interesting. Would be it overly optimistic to say that despite the open label nature of the trial, they succeeded in producing some evidence that immunosuppression is helpful for a subset? Not definitive evidence but enough to say that this line of investigation is not a dead end? Edit: Second...

The logic appears to be: not disproved, therefore valid.

There is a lack of evidence that a positive attitude enables a recovery. Arguably studies like PACE show precisely that despite infusing hope and optimism in patients, this doesn't lead to an increase activity levels or a return to work. This is not science.

What comes next?