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That's interesting. Acute dissminated encephalomyelitis is what PK Thomas diagnosed in long-term RFH patients in 1987. The rest had better beware though. He thought their symptoms "volitional". https://doi.org/10.1016/S0140-6736(87)90033-X

I think we may have been "sold a dummy". I recall previous discussions of where the idea of exercise for ME originated, given Ramsay's firm views on the subject. My recollection is that it was thought to have been used in fibromyalgia and then transferred to CFS, by analogy. This paper on Effort...

This looks to be a paper which will never stop giving. Surprising that it should have been chosen by Chalder and Butler, presumably with the approval of Wessely and David, to explain GET for CFS. We thank the participants in this study and are grateful for support from the Muscular Dystrophy...

Just realised. It was Ellen DeFreitas' retrovirus.

I just thought I would highlight this part of the Margaret Williams paper as the question of ten seems to come up about blood transfusions, some thinking it related to XMRV but most thinking it is lost in the mist of time. This appears to be the evidence but the question of what might have...

It always seemed to be a failing of reporting that in the cases of long term sequelae after the epidemics we never knew if their was any correlation between severity of initial symptoms and severity of long term chronic symptoms. Were those initially most severely affected at the RFH the ones...

I think it is instructive to se the response by Mary Sullivan in the Autumn 1989 MEA Newsletter to the article by Chalder and Butler and in particular the issues surrounding the Stokes et al paper. This broadens understanding of the matters as they were viewed at the time-before establishment of...

I'm sure TC will advise "completing the agreed activity regardless of the symptoms which occur. This is because although there is initially an increase in fatigue and other symptoms, a reduction will occur as tolerance to the activity builds up." From 1989. It still hasn't worked for me after...

It makes one wonder whether some of these doctors are at all familiar with, for example, Peter Behan's use of the term Post Viral Fatigue Syndrome which recognised all sorts of symptoms. They seem to think it was just long lasting fatigue after a virus.

That was not what Ramsay had in mind. He was thinking of something very similar to ME but without the variability of symptoms. I copied the quote to a thread the other day-but I have no idea which one.

Interesting to see the Ross Morriss quote. There is a chapter by Salkovskis and Bass on Hypochondriasis in a book on CBT by Clarke and Fairburn. They make it pretty clear that hypochondriasis and health anxiety are pretty much the same. What makes it interesting is that their oldest citation...

It does seem odd that as an infectious diseases expert he had so little knowledge of ME/CFS/PVF/PVFS. In denying the possibility of PVFS it is possible he was following the Ramsay interpretation of the post glandular fever PVFS as a constant, not variable, fatigue lasting up to 2 1/2 years. One...

OK' I can just about accept that. The problem still continues with the Wessely at als definition. By that it is closer to epidemic ME, a purely historical phenomenon, than sporadic CFS, the modern explanation. There is going to have to be a lot of reconceptualisation.

Sounds like ME for sure I'm afraid Garner is going to have to get rid of his notions of exceptionalism. His illness has a cause. CFS is defined by not having a cause. What then causes CFS? It seems astonishing that, as an infectious diseases expert, he knows of nothing, other than Dengue...

Some people are easily satisfied. Just remind me, what do they call that thing which acts as a conduit for channeling effluent?

On a recent trawl through ancient documents I came across something which is valuable supplementary evidence to the Chapter of Ramsay's book, so helpfully posted. For some reason the information seems to be omitted from the book although there was a general awareness of it. It seems...

Well, there is a difference , you know. Important people might have PVFS from covid. Not at all the sort of people who would have hysteria.

This is the point. It is perfectly understandable that someone with insufficient general experience of such cases might come up with such a hypothesis. Any reasonable person would look at the cases displaying variability of symptoms and conclude that they should look for an alternative...

It would also be helpful to have a view as to whether deconditioning could be a plausible explanation for a condition in which variability of symptoms is a major, possibly defining, feature. If deconditioning were an element of the illness- the one that put the patient to bed- how could...

What sort of funding would a study of that nature take, and what idiots would provide it?