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    Profile of circulating microRNAs in Myalgic Encephalomyelitis......(2020), Nepotchatykh, Moreau et al

    I hope they make the connection between thrombospondin, EBV duptase and ADAMTS16 gene which has been found to be „off“ in me/cfs.
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    Profile of circulating microRNAs in Myalgic Encephalomyelitis......(2020), Nepotchatykh, Moreau et al

    In case this is significant, could a treatment be as „easy“ as blocking these miRNA or us this wishful thinking?
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    Profile of circulating microRNAs in Myalgic Encephalomyelitis......(2020), Nepotchatykh, Moreau et al

    @Jonathan Edwards is this the real deal?
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    Effect of disease duration in a randomized Phase III trial of rintatolimod, an immune modulator for [ME/CFS], 2020, Mitchell et al

    still dont get it.
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    Effect of disease duration in a randomized Phase III trial of rintatolimod, an immune modulator for [ME/CFS], 2020, Mitchell et al

    what does this mean in plain?
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    Aripiprazole - Abilify

    Sounds like a garbage statement to me that 80% improve ... Unfortunately ...
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    Aripiprazole - Abilify

    Who says that and when?
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    Kynurenine pathway is altered in patients with SLE and associated with severe fatigue, 2018, Akesson et al.

    I did not want to go into the snake thing ...
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    Kynurenine pathway is altered in patients with SLE and associated with severe fatigue, 2018, Akesson et al.

    after 4 years in bed 100% I wish I was the elephant ...
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    Aripiprazole - Abilify

    what made yoz worse? ty
  11. B

    Is ME a metabolic problem or a signalling problem?

    are you aware of post polio syndromde?
  12. B

    Is ME a metabolic problem or a signalling problem?

    muscles can be damaged without high ck, like you can have mitochondrial disease without raised lactate, etc.
  13. B

    Human Leukocyte Antigen alleles associated with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): Fluge, Mella et al 2020

    which would also mean damage is most likely of permanent nature.
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    Robert Naviaux' Lab - News - from 2019 onwards

    raised extracelullar atp and mtdna are a definite in me/cfs. I have a limited understanding, that little what I have is telling me that is really bad news.
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    Pre-print: Innate immune signaling & sex differences contribute to [..] impairment, neuroinflammation, & mitochondrial rewiring in [GWI], 2020, Bryant

    ahaaa.
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    Pre-print: Innate immune signaling & sex differences contribute to [..] impairment, neuroinflammation, & mitochondrial rewiring in [GWI], 2020, Bryant

    crazy talk. people keep doing that. if you just feel fatigued, I guess you could go for a 10 k run, right? If the actual energy is no issue there is absolutely zero risk whatsoever for you to do just that. It does not matter because you just FEEL you have no energy.
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    Pre-print: Innate immune signaling & sex differences contribute to [..] impairment, neuroinflammation, & mitochondrial rewiring in [GWI], 2020, Bryant

    I simply cannot understand, and never will, why people do not understand that all of these mysterious diseases are druven by dysfunctional mitochondria. I mean in me/cfs this should be seen as quasi factious until there is 100% proof for it because it is so unbelievably obvious and everything...
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    Multi-omics Examination of Q Fever Fatigue Syndrome Identifies Similarities with Chronic Fatigue Syndrome (Preprint, 2020) Raijmakers et al.

    Hi, thank you for being here! Could you give us an update about your next steps and a timescale for these plans? Also are you looking for funding? Is there any antioxidant that you see could help in the pathology you and your colleagues found? Thank you!
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    'Holy Grail' discovery could mean 'nobody needs to worry about their MS getting worse' – study

    they should try a combination with cbt maybe!
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    A Low Glutamate Diet Improves Cognitive Functioning in Veterans with Gulf War Illness, 2020, Kirkland et al.

    it is probably not worse than a missing energy substitute for a broken tca cycle. it is absolutely and definitely no coincidence high glutamate is found in many diseases of the central nervous system. saying that, reducing glutamate in diet is probably still a good idea.
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