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    Pre-Illness Data reveals Differences in Multiple Metabolites and Metabolic Pathways in Those Who Do and Do Not Recover from IM, 2022, Jason et al

    This will turn out to b a very important paper. I can already see how people sleep on it and its vast implications. Mitochondrial dysfunction predates what people consider their trigger.
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    Open Medicine Foundation (OMF)

    How much better are you with Medtinon?
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    Red-brown speckles on palm of hands and fingers

    Did you figure this out? Having same issues!
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    Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis, Bjornevik et al (2022)

    No, it kills infected B cells. Big difference.
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    Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis, Bjornevik et al (2022)

    I think ATA 188 is/will be showing that you are wrong.
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    Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis, Bjornevik et al (2022)

    The B cells are EBV infected, that is why Rituximab probably works.
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    Plasma metabolomics reveals disrupted response and recovery following maximal exercise in ME/CFS, Arnaud Germain, Maureen R. Hanson et al, 2022

    Yes, I could send you my own metabolon results, then you could compare its results, many in itself toxic metabolites are comparable to ranges that are considered so in other diseases. Urea cycle disorders, MSUD, etc.
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    Plasma metabolomics reveals disrupted response and recovery following maximal exercise in ME/CFS, Arnaud Germain, Maureen R. Hanson et al, 2022

    Absolutely everything in this paper screams mitochondria. Also, many people underestimate how incapacitating such aberrations in specific pathways can be; Ammonia Recycling taking a little break in a PEM state? This doesn't come without a price tag.
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    Mesalazine/Mesalamine - anti-inflammatories

    Hey! Hiw severe are you? What got better, is it sustained? What has it vern prescribed for for you? Thank you.
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    Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis, Bjornevik et al (2022)

    A great many people that have been burried and ridiculed by the establishment.
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    Passive transfer of fibromyalgia symptoms from patients to mice, 2021, Goebel et al

    I am sorry but this is a nonsensical statement, it implies that there are per definition no distinct pathological conditions with a similar/same observable pattern of dysfunction in patients with similar/same symptoms. To find this pattern in 8 patients might be unlikely, but maybe still true.
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    Canada - Unknown brain disease in New Brunswick

    send prof. Garner, problem solved.
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    Selin Lab

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    Off label use of Aripiprazole shows promise as a treatment for Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) Crosby et al. 2021

    I believe Abilify could have an effect via modulating bak1. https://www.nature.com/articles/d41586-021-00429-w?fbclid=IwAR1CLhfgdBdwV4SrFIO17h8iIAsYefqMh3cUnLY5xKuzBSb5WkkBQQd1NeI I believe this (dysfunctional mitochondrial herniation) could be a big part of the me/cfs puzzle. Saying that I...
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    Aripiprazole - Abilify

    Abilify is not selling much? Ehm, no.
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    Aripiprazole - Abilify

    I think it is likely stronger than placebo, but also I believe it mediates its positive effects via dopamine and serotonine. More like rocket fuel, Abilify is.
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    Dysregulated Provision of Oxidisable Substrates to the Mitochondria in ME/CFS Lymphoblasts, 2021, Missailidis et al

    @Jonathan Edwards would you line to comment?
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    Insights from Invasive Cardiopulmonary Exercise Testing of Patients with ME/CFS, 2021, Joseph et al

    Again, most likely this is a mitochondrial issue.
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    Lipophagy confers a key metabolic advantage that ensures protective CD8A T-cell responses against HIV-1, 2020, Loucif et al

    That is because me/cfs is a mitochondrial disease.
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    [CFS]: Abnormally fast muscle fiber conduction in the membranes of motor units at a low-level static force load, Klaver-Krol et al, 2021

    Do you believe what happens in me/cfs is what is thought to be the mechanism behind post polio syndrome? „The new weakness of PPS appears to be related to the degeneration of individual nerve terminals in the motor units. A motor unit is formed by a nerve cell (or motor neuron) in the spinal...
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