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Agreed that the journal doesn't indicate 'big findings' especially as Nath delayed the whole thing by not putting it in preprint in the first place. His (mistaken) reasoning (from the get go) was that he wanted it to be in a 'top journal'.

Not so much about point scoring but gatekeeping and bias in the name of 'the real science'.

These are quite the statements by Nath, if true, especially if a pathogen is the antigen in question, I wonder how the patient commentators on this forum will react after bashing such ideas (and researchers pursuing them) for many years. Interesting to note that this comment by Nath is not...

I don't think so. The term PEM was coined in 1992 specifically for (ME/)CFS precisely because there was no existing term to describe such an unusual symptom. And Nancy Klimas is an immunologist/clinician who sees both ME and HIV/Aids patients. I'm sure she would have commented if Aids patients...

Thank you, Simon! I see it similarly, mostly. Can we be sure we are not seeing PEM in later stages of e.g. AIDS and untreated Hepatitis C, there is a good chance people would just call it "feeling bad" instead, such people simply might not have made the connection to exertion, because it's...

Hi Frenchguy, what symptom(s) prompted you to go for another MRI, what made you or the doctor reconsider and think it could be MS? I am a very severe mecfs case myself and have a very "neurological" set of symptoms. I am very sorry for everything you have to go through! Thank you!

Moved post Has there anyone somewhat qualified ever explained why the SPECT scan results by Hyde are "no evidence"?

While that is interesting, they also probably drank a lot of Sprite, that doesn't necesarily mean that's relevant to their later ME. We should definitely have a look into that though, and maybe even more importantly into why they had to take antibiotics so frequently in the first place. I am...

Prof. Hanson is fantastic! Re latent infection, I think that is a bit premature, you would expect the same "pattern" of findings in tissue damage not related to latent infection.

Nobody should be surprised about this. It will be a similar picture in ME/CFS, despite what has been said (on this forum) about (secondary) mitochondrial diseases and how they differ from what we call ME/CFS.

I can't say more about it, only that it is true with a high degree of certainty.

FWIW, the nanoneedle could not differentiate between ME and MS.

This basically means "cell damage."

While I agree that particular end organs might be of greater importance than others in ME/CFS, I think we should think more in terms of specific organelles in specific cell types, rather than whole organs. All (?) significant irregularities found in various metabollc studies can potentially be...

Hanson is the MVP in ME/CFS research, imo. Her work is very well structured and concise, very consistently so, in everything she does. It is actually fun to read most of her papers. We can be really glad to have her in our corner.

I can't know this, but from my point of POV, I think it is very likely that most patients have a mitochondrial dysfunction/disease, even if nothing suspicious was found in the first biopsy. It is expected in primary mito disease only to see what is going on after repeated biopsies

https://onlinelibrary.wiley.com/doi/10.1002/ana.26452?af=R Same old, same old? Don't think so.

I think the OMF does excellent work. I think Ron has done excellent work. You see what I am saying? We owe Ron, like the OMF does, that doesn't automatically imply giving him more money is the best way forward.

Great scientist, great man, community has to move on...

Bunch of bullshit.