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My guess is that titles are not written by the authors, this is media related. This often happens even on Youtube as well. It may be editors, it may be someone else, but its done to try to maximise readership. Headlines are not about accuracy.

My guess is it might be funding related. They might have been told that funding was more likely to be approved if they used Fukuda. It would be nice to find out for sure though.

There have been two large studies so far, which showed distinct issues in both ME/CFS and Lyme patients, with substantial overlap. I do not recall the details, maybe someone else can comment?

Yes.

In the short term yes, in the long term no. The body then compensates by increasing oxygen dumping, due to changes in enzyme synthesis. The short term changes in acid-base balance are compensated for by long term changes in enzyme synthesis. I am unsure what long term effects prolonged...

Actual lactic acidosis is usually fatal without treatment. Elevated lactic acid is not the same thing, but can become lactic acidosis if it persists. However our lower than normal oxygen utilisation might confer some resistance to the problem. I do know we get elevated lactate, but it has to be...

You should talk about it with your doctor, but I am not aware of any ME patient who was in acidosis. I think the biochemistry of ME can induce all the same symptoms. Newton in the UK showed that in her patient group we are actually alkaline except when exerting ourselves and for a fair while...

This was my primary disabling symptom, but over far more of my body than just my hands, for nearly twenty years. I found repetitive muscle use was a problem, but static muscle stress was actually far worse. I found pacing those activities was the only thing that really worked over the long term...

I am not aware of any evidence of increased RBC or erythropoietin in ME patients. The early response of local oxygen problems is an increase in lactic acid, and usually this is in the presence of lots of carbonic acid. This acidification causes RBCs to dump more oxygen due to enzymatic effects...

I had over fifteen years of severe pain and for most of it took nothing. Because nothing worked. Pacing helped the most, by not aggravating things. Vioxx, which was banned due to vascular risks, I took as occasional help, and it worked, but I was never game to take it regularly as I was well...

Most of which are never tested for, mostly because there are no tests for most of them. We keep discovering new ones too.

Exactly.

This is often overlooked. We need at least the basics on spotting bad research, because it will be thrown at us as "proof". Its not just psychosocial research either.

Its worse than that for much of the world. In any system with private insurance, or even government workers compensation schemes, failure to disclose is grounds to deny further rights.

While speculative, this could be a potential link between our "something in the blood" and possible brain changes.

This is interesting but not the paper I was referring to. I was referring to the paper identified by David Tuller in a thread on this forum. It is specifically on SD and SF36PF in CFS. I do not recall the exact name or reference.

This option. SD is undefined on this kind of dataset. PDW wrote a paper in 2007 showing he understood it was a biased measure, one which in fact biases toward their hypothesis. Look up his paper on SD for SF36PF data. This is deliberate manipulation of data to obtain a answer biased toward their...

I was wondering how long it would take for someone to comment. ;)

Fraud and Charcot started this, in the nineteenth century. Knowledge of disease was minimal. By the early to mid twentieth century it was well established. I am unsure at what point it was first taught in medical schools, maybe someone can tell us, but after that all doctors were exposed to the...

Sadly, that about sums it up, though its more soft whispering in the right ears.