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Interesting that this is very similar to the Canadian census data from 2014, which found 1.4% of adults reporting CFS, with a similar breakdown between men and women as well (1.0% versus 1.7%). The Canadian survey also asked specifically about diagnosis by a health professional as this US ones does.

Looks like this is moderate and severe Covid patients, i.e. hospitalized. They include those who died and presumably would have been diagnosed while in hospital for Covid.

In the US, traffic deaths were significant higher in 2020 than in previous years (and then higher again in 2021). So it wasn't a dip in 2020 with a rebound in 2021. I don't think it is well understood why (though part of it is that with less traffic some people tend to drive like complete...

The paper reports a correlation between cumulative covid cases and road deaths (and no significant correlation with past month covid cases and road deaths), which doesn't suggest that it was acute covid. I don't know that I find the paper that convincing, but alongside the other papers we've...

I think it would be mannan-binding lectin deficiency (which is very common).

It was pointed out to me that the answer to this is in the figures:

Abstract Objective To examine data on COVID-19 disease associated with a 10 percent increase in U.S. road deaths from 2020 to 2021 that raises the question of the potential effect of pandemic stress and neurological damage from COVID-19 disease. Methods Poisson regression was used to estimate...

Maybe it would be interesting to look at gut microbiota or gut symptoms and LBP in those with high levels.

Not really clear how they are separating patients who they say do or don't have immunodeficiency: But even with this somewhat mysterious stratification, the differences they are showing do not look very significant: Claiming there is a difference between groups here seems like a stretch...

Are mothers with ME more likely to have children with ME than fathers with ME are? I've always wondered about this, partly because it is true in my own family, and have seen others suggest it might be true (most recently Fereshteh Jahaniani in the NIH genetics webinar), but I can't seem to find...

This seems very confused. Their definition of having persistent symptoms is: This looks very broad, so of course it would be associated with baseline symptoms. And as @sneyz says, the baseline isn't even a baseline, it's while they are receiving antibiotic treatment for acute Lyme. So people...

They do actually report vaccine effectiveness by variant (Pre-alpha/Alpha, Delta, Omicron): 41% vaccine effectiveness versus LC after Omicron is a lot less than 58%.

The difference could also be lower rates of LC from Omicron versus pre-Omicron variants (the vaccinated cohort, on the right, was 75% Omicron, while the unvaccinated cohort was 88% pre-Omicron). That's definitely something we've seen in other studies.

This seems like it might just be a multiple comparisons problem, because they checked if a whole lot of things were correlated and found a few closely related things were, which is not surprising. Their analysis is not that there is a correlation between the neutralization of BA.5 as a...

Yes, this one is definitely hopeless. If you look at their risk ratios, it's pretty clear that there is some kind of selection problem with their infected versus negative cohorts. Sure looks like the infected cohort is somehow healthier and having fewer symptoms than the negative cohort (aside...

They were awarded a grant for the Raman microscope they wanted to continue this research:

One wonders how you could do a study like this and not ask the participants if they have LC, which seems like the most important question here.

I think the fact that they call chronic fatigue syndrome a psychiatric condition tells us all we need to know. Not to mention that their internists apparently thought more than half of the patients in the study, who were at an LC clinic, had excellent or good physical health.

That's the part I don't understand. If Nath is saying he thinks it is persistent antigen, but not active infection, then it pretty much has to be autoimmune, because otherwise it makes no sense for people to stay ill for decades. I guess you can imagine some kind of repeated infection or maybe...

I guess we'll get a real answer to this one in a couple years when the Brazilian Fluvoxamine study reports out (1500 patients in control / Fluvoxamine / Metformin, though I'm not sure 60 (+30 & 90) day FSS improvement is a great outcome).