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I have been focusing on the a7 nicotine recepter but there is more to it. Mammals make lynx1.

http://www.informatics.jax.org/allele/MGI:3716630 J:122965 Miwa JM, et al., The prototoxin lynx1 acts on nicotinic acetylcholine receptors to balance neuronal activity and survival in vivo. Neuron. 2006 Sep 7;51(5):587-600

http://www.informatics.jax.org/allele/MGI:3716630 The LYNX1 gene encodes a protein that shares characteristics with toxins that bind and inhibit nicotinic acetylcholine receptors (nAChR; see, e.g., CHRNA1, 100690) (Miwa et al., 1999). Components of snake venoms often have structural and...

https://www.cell.com/neuron/fulltext/S0896-6273(11)00254-6?_returnURL=https%3A%2F%2Flinkinghub.elsevier.com%2Fretrieve%2Fpii%2FS0896627311002546%3Fshowall%3Dtrue This review discusses three emerging hypotheses about ways that the nicotinic system can be modulated. First is the role played by...

It docks at the allosteric site of the a7 nicotine receptor. These receptors are on many cells. It has many effects.

The med I use is an a7nachr modulator.

Looking at what happened with the animals after the mysterious chronic infection was brought in I would say look at the neurotransmitter achetylcholine and receptors. As the allosteric a7nicotine achetylcholine receptor modulator was the med to treat the animals with and it also saved my live...

They probebly are right but looking at the wrong substance.

No.

Review ARTICLE Front. Neurol., 10 January 2019 | https://doi.org/10.3389/fneur.2018.01033 Neuroinflammation and Cytokines in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): A Critical Review of Research Methods Michael B. VanElzakker*, Sydney A. Brumfield and Paula S. Lara Mejia...

https://bmcmedgenomics.biomedcentral.com/articles/10.1186/s12920-017-0248-3 Epigenetic modifications and glucocorticoid sensitivity in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS)

Another class of lipids with farreaching activity on virtually all organ systems are steroids; androgenic, progestin, and corticosteroids are broadly reduced in our patient cohort. https://ammes.org/tag/dexamethasone/

https://www.genengnews.com/news/bile-acid-metabolites-keep-the-guts-t-cells-in-good-humor/ Bile Acid Metabolites Keep the Gut’s T Cells in Good Humor https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6099188/ Bile Acids Activated Receptors Regulate Innate Immunity...

Me too

They remove the whole glands. Look at all the things you have to compensate for.

Adrena glands have a7nach receptors.

Yes, but I do not see what it can tell. Why create an Addison-patiënt? Addison is very difficult to control.

Yes, but what will that tell????

Cut, cut, cut. NO WAY. Why would you want to activate astrocytes and transforming growth factor beta (TGF-β) ????? Ain't astrocytes not already activated in ME patients?

Bile is involved. A greenish glow over the fresh stool was one of two symptoms that together with a dry cough told me the animals had a flare and needed levamisole. The stools also looked as if the fat was not properly digested.